Maria Lampinen
Forskare vid Institutionen för medicinska vetenskaper; Gastroenterologi/hepatologi
- Telefon:
- 018-611 41 97
- Mobiltelefon:
- 070-867 05 20
- Fax:
- 018-611 37 03
- E-post:
- Maria.Lampinen@medsci.uu.se
- Besöksadress:
- Akademiska sjukhuset, Ingång 40, 5 tr
751 85 Uppsala - Postadress:
- Akademiska sjukhuset, Ingång 40, 5 tr
751 85 UPPSALA
Forskare vid Institutionen för farmaci; Immunonkologi
- Mobiltelefon:
- 070-867 05 20
- E-post:
- maria.lampinen@farmaci.uu.se
- Besöksadress:
- Biomedicinskt Centrum BMC, Husargatan 3
752 37 Uppsala - Postadress:
- Box 580
751 23 UPPSALA
Ladda ned kontaktuppgifter för Maria Lampinen vid Institutionen för farmaci; Immunonkologi
- Akademiska meriter:
- Docent
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Publikationer
Senaste publikationer
- Phenotypic Switch and Growth of Melanoma Spheroids in the Presence of Mast Cells (2023)
- Fecal Biomarkers of Neutrophil and Eosinophil Origin Reflect the Response to Biological Therapy and Corticosteroids in Patients With Inflammatory Bowel Disease (2023)
- Monensin induces secretory granule-mediated cell death in eosinophils (2023)
- Mefloquine causes selective mast cell apoptosis in cutaneous mastocytosis lesions by a secretory granule-mediated pathway (2022)
- Mast cell chymase affects the functional properties of primary human airway fibroblasts (2022)
Alla publikationer
Artiklar
- Phenotypic Switch and Growth of Melanoma Spheroids in the Presence of Mast Cells (2023)
- Fecal Biomarkers of Neutrophil and Eosinophil Origin Reflect the Response to Biological Therapy and Corticosteroids in Patients With Inflammatory Bowel Disease (2023)
- Monensin induces secretory granule-mediated cell death in eosinophils (2023)
- Mefloquine causes selective mast cell apoptosis in cutaneous mastocytosis lesions by a secretory granule-mediated pathway (2022)
- Mast cell chymase affects the functional properties of primary human airway fibroblasts (2022)
- Monensin induces selective mast cell apoptosis through a secretory granule-mediated pathway. (2021)
- Mast Cell β-Tryptase Is Enzymatically Stabilized by DNA (2020)
- Genetic and shared environmental risk factors do not lead to eosinophil activation in healthy twins of IBD patients (2020)
- Prognostic significance of faecal eosinophil granule proteins in inflammatory bowel disease (2019)
- High serum sCD40 and a distinct colonic T cell profile in ulcerative colitis associated with primary sclerosing cholangitis. (2019)
- Tissue factor in ulcerative colitis, with and without concomitant primary sclerosing cholangitis. (2019)
- Prognostic significance of eosinophil granule proteins in inflammatory bowel disease (2018)
- Downregulated eosinophil activity in ulcerative colitis with concomitant primary sclerosing cholangitis (2018)
- Clinical implications of assay specific differences in f-calprotectin when monitoring inflammatory bowel disease activity over time (2017)
- Siramesine causes preferential apoptosis of mast cells in skin biopsies from psoriatic lesions (2017)
- A key requirement for CD300f in innate immune responses of eosinophils in colitis (2017)
- Expression of the liver homing receptor CXCR3+on colonic CD8+T lymphocytes in patients with primary sclerosing cholangitis provides a possible link between colonic and biliary duct inflammation (2016)
- Evaluation of biomarkers for ulcerative colitis comparing two sampling methods (2016)
- Ablation of human skin mast cells in situ by lysosomotropic agents (2015)
- Naive T cells in the gut (2014)
- Naive T cells correlate with mucosal healing in patients with inflammatory bowel disease (2014)
- CD14(+)CD33(+) myeloid cell-CCL11-eosinophil signature in ulcerative colitis (2013)
- Increased Fecal Levels of Chromogranin A, Chromogranin B and Secretoneurine in Collagenous Colitis (2013)
- PI3-Kinase Regulates Eosinophil and Neutrophil Degranulation in Patients with Allergic Rhinitis and Allergic Asthma Irrespective of Allergen Challenge Model (2012)
- C3b-induced eosinophil degranulation involves PI3-kinases and is inhibited by protein kinase C activity (2011)
- Patients with allergic rhinitis and allergic asthma share the same pattern of eosinophil and neutrophil degranulation after allergen challenge (2011)
- Fecal eosinophil cationic protein as a marker of active disease and treatment outcome in collagenous colitis (2011)
- Eosinophils Express Muscarinic Receptors and Corticotropin-Releasing Factor to Disrupt the Mucosal Barrier in Ulcerative Colitis (2011)
- Budesonide Treatment of Patients With Collagenous Colitis Restores Normal Eosinophil and T-cell Activity in the Colon (2010)
- Intestinal Macrophage/Epithelial Cell-Derived CCL11/Eotaxin-1 Mediates Eosinophil Recruitment and Function in Pediatric Ulcerative Colitis (2008)
- Different regulation of eosinophil activity in Crohn's disease compared with ulcerative colitis (2008)
- Accumulation, activation and survival of neutrophils in ulcerative colitis (2008)
- Albumin stimulation of eosinophil migration involves PI3-kinases and is associated with diminished eosinophil CD49d and CD49f expression. (2006)
- Selective priming of peripheral blood eosinophils in patients with idiopathic hypereosinophilic syndrome (2006)
- Eosinophil granulocytes are activated during the remission phase of ulcerative colitis. (2005)
- Cytokine-regulated accumulation of eosinophils in inflammatory disease (2004)
- IL-5 and TNF-alpha participate in recruitment of eosinophils to intestinal mucosa in ulcerative colitis. (2001)
- Interleukin-2 inhibits eosinophil migration but is counteracted by IL-5 (2001)
- The role of interleukin-5, interleukin-8 and RANTES in the chemotactic attraction of eosinophils to the allergic lung (see comments) (1999)
- Identification of IL-5 and RANTES as the major eosinophils chemoattractants in the astmatic lung (1996)
- Intravenous anti-P. aeruginosa IgY antibodies increase bacterial concentration in porcine septic shock with bacteremia
